Franco J. Vaccarino
Name: Franco J.
Surname: Vaccarino
Section: Dr. Franco J. Vaccarino has been president and vice-chancellor of the University of Guelph (U of G) since 2014. ,CURRENT POSITION Vice Chancellor/President, University of Guelph
Qualification: Before joining the U of G, Dr. Vaccarino served since 2007 as principal of the University of Toronto Scarborough (UTSC) and vice-president of the University of Toronto. He joined UTSC’s Department of Psychology in 1984, and ultimately served as its chair. In Toronto, he was graduate chair of the tri-campus Graduate Department of Psychology and head of the Department of Psychiatry’s neuroscience program.
Education and career
Dr. Vaccarino’s studies of the neurobiology of stress and motivation are considered a model for bridging the neural and behavioural sciences. An internationally recognized researcher and teacher, he has authored more than 100 academic publications, and is a sought-after international speaker on the neurobiology of substance abuse and brain changes from alcohol and drug dependency. In 2015, he was one of two principal editors of a national report on the effects of early and frequent marijuana use during adolescence released by the Canadian Centre on Substance Abuse. Dr. Vaccarino has been honoured by the Canadian Institutes of Health Research and the Canadian College of Neuropsychopharmacology. In 2014, he was named a Fellow of the Canadian Academy of Health Sciences, considered a top honour for individuals in health sciences in Canada. He has served as executive vice-president (programs) and vice-president (research) at the Centre for Addiction and Mental Health, and as vice-president and director of research, Clarke Institute of Psychiatry, both in Toronto. Dr. Vaccarino holds a BSc from the University of Toronto and an MSc and a PhD in psychology from McGill University, and was a post-doctoral researcher at the Scripps and Salk institutes in California.
Research focus
1，Kennedy S H, Evans K R, Krüger S, et al. Changes in regional brain glucose metabolism measured with positron emission tomography after paroxetine treatment of major depression[J]. American Journal of Psychiatry, 2001, 158(6): 899-905. 2，Swerdlow N R, Vaccarino F J, Amalric M, et al. The neural substrates for the motor-activating properties of psychostimulants: a review of recent findings[J]. Pharmacology Biochemistry and Behavior, 1986, 25(1): 233-248. 3，Kennedy S H, Javanmard M, Vaccarino F J. A review of functional neuroimaging in mood disorders: positron emission tomography and depression[J]. The Canadian Journal of Psychiatry, 1997, 42(5): 467-475. 4，Vaccarino F J, Bloom F E, Koob G F. Blockade of nucleus accumbens opiate receptors attenuates intravenous heroin reward in the rat[J]. Psychopharmacology, 1985, 86(1-2): 37-42. 5，Vaccarino F J, Bloom F E, Koob G F. Blockade of nucleus accumbens opiate receptors attenuates intravenous heroin reward in the rat[J]. Psychopharmacology, 1985, 86(1-2): 37-42. 6，Javanmard M, Shlik J, Kennedy S H, et al. Neuroanatomic correlates of CCK-4-induced panic attacks in healthy humans: a comparison of two time points[J]. Biological psychiatry, 1999, 45(7): 872-882. 7，Evans K R, Vaccarino F J. Amphetamine-and morphine-induced feeding: evidence for involvement of reward mechanisms[J]. Neuroscience & Biobehavioral Reviews, 1990, 14(1): 9-22. 8，Vaccarino F J, Rankin J. Nucleus accumbens cholecystokinin (CCK) can either attenuate or potentiate amphetamine-induced locomotor activity: evidence for rostral-caudal differences in accumbens CCK function[J]. Behavioral neuroscience, 1989, 103(4): 831.